<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Baird, Andrew</style></author><author><style face="normal" font="default" size="100%">Costantini, Todd</style></author><author><style face="normal" font="default" size="100%">Coimbra, Raul</style></author><author><style face="normal" font="default" size="100%">Eliceiri, Brian P</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Injury, Inflammation and the Emergence of Human Specific Genes</style></title><secondary-title><style face="normal" font="default" size="100%">Wound Repair and Regeneration</style></secondary-title></titles><dates><year><style  face="normal" font="default" size="100%">2016</style></year></dates><urls><web-urls><url><style face="normal" font="default" size="100%">http://onlinelibrary.wiley.com/doi/10.1111/wrr.12422/abstract</style></url></web-urls></urls><publisher><style face="normal" font="default" size="100%">Wiley Online Library</style></publisher><isbn><style face="normal" font="default" size="100%">1524-475X</style></isbn><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p&gt;Abstract In light of the central role of inflammation in normal wound repair and regeneration, we hypothesize that the preponderance of human-specific genes expressed in human inflammatory cells is commensurate with the genetic versatility of inflammatory response ...&lt;/p&gt;
</style></abstract><notes><style face="normal" font="default" size="100%">&lt;p&gt;http://onlinelibrary.wiley.com/doi/10.1111/wrr.12422/abstract&lt;/p&gt;
</style></notes></record></records></xml>