<?xml version="1.0" encoding="UTF-8"?><xml><records><record><source-app name="Biblio" version="7.x">Drupal-Biblio</source-app><ref-type>17</ref-type><contributors><authors><author><style face="normal" font="default" size="100%">Riesenfeld, A</style></author></authors></contributors><titles><title><style face="normal" font="default" size="100%">Endocrine control of skeletal robusticity.</style></title><secondary-title><style face="normal" font="default" size="100%">Acta Anat (Basel)</style></secondary-title><alt-title><style face="normal" font="default" size="100%">Acta Anat (Basel)</style></alt-title></titles><keywords><keyword><style  face="normal" font="default" size="100%">Animals</style></keyword><keyword><style  face="normal" font="default" size="100%">Bone and Bones</style></keyword><keyword><style  face="normal" font="default" size="100%">Bone Development</style></keyword><keyword><style  face="normal" font="default" size="100%">Castration</style></keyword><keyword><style  face="normal" font="default" size="100%">Depression, Chemical</style></keyword><keyword><style  face="normal" font="default" size="100%">Diet</style></keyword><keyword><style  face="normal" font="default" size="100%">Endocrine Glands</style></keyword><keyword><style  face="normal" font="default" size="100%">Estrogens</style></keyword><keyword><style  face="normal" font="default" size="100%">Female</style></keyword><keyword><style  face="normal" font="default" size="100%">Hypophysectomy</style></keyword><keyword><style  face="normal" font="default" size="100%">Male</style></keyword><keyword><style  face="normal" font="default" size="100%">Organ Size</style></keyword><keyword><style  face="normal" font="default" size="100%">Ovary</style></keyword><keyword><style  face="normal" font="default" size="100%">Pituitary Gland</style></keyword><keyword><style  face="normal" font="default" size="100%">Progesterone</style></keyword><keyword><style  face="normal" font="default" size="100%">Rats</style></keyword><keyword><style  face="normal" font="default" size="100%">Sex Factors</style></keyword><keyword><style  face="normal" font="default" size="100%">Stimulation, Chemical</style></keyword><keyword><style  face="normal" font="default" size="100%">Testis</style></keyword><keyword><style  face="normal" font="default" size="100%">Testosterone</style></keyword><keyword><style  face="normal" font="default" size="100%">Thyroid Gland</style></keyword><keyword><style  face="normal" font="default" size="100%">Thyroidectomy</style></keyword></keywords><dates><year><style  face="normal" font="default" size="100%">1975</style></year><pub-dates><date><style  face="normal" font="default" size="100%">1975</style></date></pub-dates></dates><volume><style face="normal" font="default" size="100%">91</style></volume><pages><style face="normal" font="default" size="100%">481-99</style></pages><language><style face="normal" font="default" size="100%">eng</style></language><abstract><style face="normal" font="default" size="100%">&lt;p&gt;Changes of skeletal robusticity result from a mosaic response of various parts of the skeleton to given endocrine changes. The character of these reactions corresponds to that of muscles which act as selective target areas for specific hormones. Robusticity changes are mediated by increases or decreases of the length and weight of the bones. The changes are probably species- or even subspecies-specific.&lt;/p&gt;
</style></abstract><issue><style face="normal" font="default" size="100%">4</style></issue><custom1><style face="normal" font="default" size="100%">&lt;p&gt;http://www.ncbi.nlm.nih.gov/pubmed/1154991?dopt=Abstract&lt;/p&gt;
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